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The striavascularis in the aged, null mice was thinner than in the heterozygous or wildtype mice.
2
Cells expressing Sox10 were present in the striavascularis, outer sulcus and spiral prominence in mouse and human cochleas.
3
GR receptor mRNAs were demonstrated neither in cells of the striavascularis nor in cells of the organ of Corti region.
4
Immunoreactivity for sPLA2 also was detected in cultured marginal cells of the striavascularis and in fibrocytes of the spiral ligament.
5
We observed degeneration of striavascularis intermediate cells, the cells that generate the endocochlear potential, but no other abnormalities within the cochlea.